Appreciably elevated anti-EBOV IgG antibody titres in survivors with arthralgias in comparison with survivors without arthralgias assist a task for sustained immune activation and inflammation in the pathogenesis of arthralgias soon after EVD229. This hypothesis is consistent with conclusions of polyarthropathies connected to other viral infections

The latest studies have confirmed that although EBOV has long been regarded immunosuppressive, EBOV-certain cellular and humoral immune responses create but are sometimes outpaced during the host–pathogen EVD ‘arms race’122,123, where timing appears essential. This host–pathogen competition may additionally be applied to vaccine-mediated me

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Article Under Review